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Description
Cytotoxic activity of tumor necrosis factor is mediated by early damage of mitochondrial functions
The IL-17 secreted by these cells binds to IL-17 receptors (IL-17Rs) expressed in midbrain neurons, inducing neuronal death through the upregulation of NF-B and downstream signaling pathways, as demonstrated in human iPSC-derived neuronal models
RSL3), ***p = 0.0009 (RSL3 vs
SCFAs, especially butyrate, play a central role in immune regulation
