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Ultimately, ferroptotic microglia release damage-associated molecular patterns (DAMPs), including free iron, oxidized phospholipids, and HMGB1 ( 5.3.1.2 Microglia and AD The pathological significance of activated microglia in the brains of AD patients was first systematically described by McGeer and colleagues in 1987, a discovery that ignited extensive investigation into their potential role in neurodegenerative progression

Cagrilintide, the second generation (2020s) Novo Nordisk took the Pramlintide concept and applied the same half-life extension strategy that they used with Semaglutide: Kept the proline substitutions (anti-amyloid) Added a C20 fatty diacid via a -Glu linker to a lysine residue, albumin binding Additionally stabilized the N- and C-terminal positions Result: Cagrilintide with a half-life of 6.6 days (vs 50 minutes for Pramlintide)

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