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Description
Research by Dhamija et al

Data are mean s.d

335 As documented in this section and Table 3, antineoplastic agents/methods with ROS-inducing potential and their ROS-mediated antitumor mechanismscompiled from sources including the FDA database (www.fda.gov), ClinicalTrials.gov, and DrugBank ( diverse mechanisms of action ranging from metabolic interference to direct oxidative damage

In this review, the role of glutathione and glutathione-dependent enzymes in the maintenance of redox homeostasis is summarized
