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Each unique mutation has a different impact on how the MTHFR enzyme performs within our body

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PLoS Pathog

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suggest that the pathogenesis of mitochondrial-associated epilepsy involves processes such as neuronal energy depletion, oxidative stress, impaired calcium signaling, neurotransmitter imbalances, -oxidation deficiency, and glial cell dysfunction (3)

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This can lead to neurodegenerative diseases such as Alzheimers and Parkinsons

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