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Case is strong and

Lp(a) can thus compete with plasminogen for fibrin binding (inhibiting fibrinolysis) and contribute to the formation of endothelial clots, i.e., atherogenic plaques ( Another role for Lp(a) has also been hypothesized, following on the observations that 90% of oxidized lipoproteins (oxLPs) bound to ApoB-containing lipoproteins are actually bound to Lp(a) and that LP-PLA2, an enzyme that degrades atherogenic oxLPs, is associated with Lp(a)

Lipid and lipoprotein metabolism

Brown, Jr
