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Description
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This is driven by the likelihood of subchondral bone changes as a consequence of failed repair, particularly if the first attempt involved disruption of the tidemark for marrow recruitment
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Many of these neuroanatomical areas are responsible for cognition, motor function, and affect [4]
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Moreover, tamoxifen-induced JNK activation was not reversed by inhibiting ASK1 (Apoptosis signal regulating kinase, Supplementary Fig
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