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Shen et al
This inflammatory status in the CNS is evidenced by the presence of oxidative stress, increased production and release of pro-inflammatory mediators and the development and progression of neurodegenerative pathologies (von Bernhardi et al., 2015)
class D ESBLs, such as OXA-2 and OXA-10-like enzymes
Deficiency of canonical Wnt/-catenin signalling in hepatic dendritic cells triggers autoimmune hepatitis
