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However, current evidence suggests that the disorder arises from a complex interplay of genetic predisposition, altered monoamine neurotransmission, and structural brain abnormalities, which together increase susceptibility
Eriksson CJ
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10.1016/j.colsurfb.2018.08.001 108 RamaziS.DaddzadiM.SahafnejadZ.AllahverdiA
