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By targeting iron-dependent lipid peroxidation, ferroptosis induction bypasses key resistance pathways associated with chemotherapy, radiotherapy, and immunotherapy, while exploiting glioblastomas metabolic vulnerabilities

Due to its low molecular weight (2.016 g/mol), H 2 can rapidly diffuse across cell membranes, penetrating the stratum corneum and reaching subcellular compartments such as mitochondria and nuclei, which mitigates oxidative stress at its source [6,7]

Pro-oxidants are reactive oxygen species that increase oxidative damage and inflammation

Antimalarial drugs such as pamaquin are oxidizing compounds that deplete reduced glutathione