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Antioxidants protect your cells from oxidative stress and genetic damage

Under zinc-deficient conditions, mutant SOD1 can generate highly active hydroxyl radicals and peroxynitrite via abnormal copper chemistry, further exacerbating neuronal oxidative damage and protein nitration (120)

After activation of the TGF pathway in PSCs cultured in low glucose, the expression levels of -SMA and collagen I were significantly upregulated

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