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Activation of Tyrosine Kinase C-Abl Contributes to -Synuclein-Induced Neurodegeneration

In addition to their potentially positive actions on toning down inflammatory signaling, all three peptides have been posited to also support cellular regeneration via different mechanisms that ultimately support vascularity and nutrient delivery to the cellular structure

This article is part of a Special Issue entitled: Metals in Bioenergetics and Biomimetics Systems

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