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Two histologic endpoints dominate MASH drug development: resolution of steatohepatitis without worsening of fibrosis , and improvement in fibrosis by at least one stage without worsening of steatohepatitis
Cell size and invasion in TGF-beta-induced epithelial to mesenchymal transition is regulated by activation of the mTOR pathway
The complexity of their subunit composition 25 leads to highly diverse functional and pharmacological properties, enabling the sensitive regulation of synaptic transmission 2,27,28,29,30,31,32,33,34,35,36
[PubMed] [Google Scholar] 44.Elliott G., Whited B.A., Purmalis A., Davis J.P., Field S.O., Lancaster C., Robert A
