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Such a setup would precisely delineate the NSC-derived paracrine factors (e.g., BDNF, VEGF [18, 32], TGF-) responsible for suppressing microglial activation and promoting an anti-inflammatory phenotype, independently of in vivo complexities
[(accessed on 27 June 2020)]
Inhibition of Neuronal Ferroptosis Protects Hemorrhagic Brain
