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6 Conclusion and outlook The intricate ferroptosis-immune-metabolic axis has emerged as a central driver of COPD pathogenesis, creating a self-perpetuating cycle of inflammation, oxidative stress, and tissue damage
Recent studies have found that calycosin inhibited tMCAO/R or OGD/R-induced ACSL4 upregulation and promoted the recovery of neural function after cerebral ischemia in rats (Liu et al
Author contributions Conceptualization, M.F