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Excessive intracellular free radicals inhibit the activation of protein kinase Avia (PKA) oxidative modification of thiol groups in cysteine residues, while a large amount of ROS released during mitochondrial oxidative stress could further exacerbate this inhibition [20], IDB protected PKA from oxidative damage by suppressing the generation and accumulation of mitochondrial ROS [36]
However, the Cochrane review conducted by Showell et al
Hu C, Liu JY, Wang W, Mota-Sanchez D, He S, Shi Y
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