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Meanwhile, higher curcumin dosage induced mitochondrial and nuclear DNA damages to human hepatoma G2 cells whereby lipid peroxidation and ROS levels were markedly elevated under conditions of high curcumin doses [266]
Similarly, treatment with GSH synthesis inhibitors or GPX4 inhibitors can impact the regenerative response after axon injury in a dose-sensitive manner, with a low dose promoting axonal fusion and a high dose causing axonal debris
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