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The CPT1C 5 UTR contains a repressing upstream open reading frame that is regulated by cellular energy availability and AMPK
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Emerging evidence suggests that ACs play a pivotal role in immune-metabolic pathways, linking lipid metabolism to mitochondrial function, with inadequate energy production contributing to metabolic stress, inflammation, and insulin resistance, all of which tied to depression pathophysiology [15]
Inhibition of lipid oxidation increases glucose metabolism and enhances 2-deoxy-2-[(18)F]fluoro-d-glucose uptake in prostate cancer mouse xenografts
