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Redox imbalance, FeRD in particular, results from virus-induced HMRD and represents a critical state both in the pathogenesis of SARSCoV2 infection and host inflammatory response 27,48,49

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the remaining one-quarter is synthesized in the human body from lysine (creating carnitine carbon skeleton) and methionine (origin of N-methyl groups) with the participation of ascorbic acid, niacin, pyridoxine, and Fe +2 [53] in the liver, kidneys, brain [10], and placenta [54]