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The adaptor molecule MyD88 probably acts as a bridge between the innate and the adaptive immunity, by promoting the activation of antigen presenting cells and consequently priming lymphocytes that exhibit IFN- secretion and cytotoxicity profile against tumor cells

Metabolic profiles of cardiomyocytes stimulated by 2-AB imply the activation of the serine biosynthesis pathway (Fig

10.1155/2017/9747256 Biomed Res Int