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Description
Mutations in the APOB gene represent about 1%5% of FH cases, whereas mutations in the PCSK9 gene represent no more than 3% of identified cases of FH
CRP can negatively modulate NO production and can lead to some other procoagulant effects, such as reducing PGI2 release, diminishing fibrinolysis, the release of tissue factor, and the increase in thrombocyte adhesiveness
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