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Notably, obesity disrupts both adipose NAD + homeostasis and sirtuin enzymatic function, leading to mitochondrial deficits and metabolic complications (411)

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Taken together, the action mechanisms of AFB1-exposure-induced mitochondrial dysfunction may include direct damage to the mitochondrial ultrastructure, a decrease in mitochondrial membrane potential, inhibition of TCA cycle-rate-limiting enzymes and mitochondrial biogenesis, loss of ETC complex activities, and a decrease in ATP levels

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