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The mechanisms by which PPAR loss leads to aberrant apoptosis and reduced cell survival have been associated to altered lipid metabolism and accumulation of stress and pro-inflammatory molecules such as reactive lipids and ROS deriving from the 5-LO and COX2 pathways, that specifically activate a lipid-mediated programmed cell death [33]

For example, the inhibition of FAO inhibited paclitaxel-resistant lung adenocarcinoma cell proliferation 34 and increased the resistance of chronic lymphocytic leukemia to glucocorticoids

doi: 10.1016/j.bbamem.2006.09.024 125