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Furthermore, Pb-induced dysfunction of synapsin and synaptophysin not only suggests compromised vesicle mobilization from reserve pools but also points to impaired docking at the active zone [103,106]
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Secondary metabolites from gentiana cruciata l
This suggests that AOD 9604 may potentially augment the anti-cancer potency of doxorubicin while possibly minimizing unintended actions associated with non-target tissue exposure
