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found that cytosolic aspartate aminotransaminase (GOT1) inhibition represses mitochondrial metabolism and enhances labile iron availability through autophagy, which accelerates pancreatic cancer cell death by ferroptosis (Kremer et al., 2021)

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endothelial NOS (eNOS), which is initially identified in vascular endothelial cells

For those serving the research community, alternative platforms and high-risk processing remain necessary
