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Autophagy in cisplatin ototoxicity In the study of cisplatin resistance, it was found that cancer cells can reduce cisplatin therapy efficacy by triggering autophagy and autophagy-modulating agents as chemosensitizers for cisplatin therapy in cancer

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Pathological -synuclein transmission initiated by binding lymphocyte-activation gene 3

This substantial participant database provides meaningful safety evidence across varied dosing regimens, administration routes (oral and injectable), and treatment durations, making it one of the more extensively studied peptide fragments in human clinical trials