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Additionally, it has been shown that the injection of amrinone at a dose of 5 mg/(kgday 1 ) for 5 days in septic rats helped to induce the activation of PDHC in skeletal muscle and to significantly reduce the level of lactate, suggesting that amrinone may be useful for the treatment of hyperlactatemia in septic patients (64)

Further research is warranted to investigate differences of p-wave characteristics in critically ill patients who do and those who do not develop NOAF

Tau malfunction causes a defect in this transport mechanism leading to cytotoxicity and neurodegeneration as they can propagate and induce toxicity in other cells [2,3,4]

doi: 10.1017/neu.2015.10