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Specifically, these exposures can alter mitochondrial redox homeostasis and oxidatively damage DNA [106, 107], RNA [106], lipids, and proteins, through PTMs [8, 26, 48, 70, 108, 109], causing inactivation of their functions and signaling pathways, leading to mitochondrial dysfunction [26, 53, 68, 83], death of hepatocytes, and liver damage [17, 110] or age-related conditions [91, 111,112,113]