glutathione increases in cancer cells metabolism progression and treatment resistance Glutathione-Dependent Pathways in Cancer Cells
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The transient overexpression of transcription factor LcERF107 demonstrated the transcriptional regulation of glutathione metabolism pathway genes and its ability to increase the activities of antioxidant enzymes such as CAT, APX, SOD and POD to increase the scavenging capacity of reactive oxygen species to improve the tolerance to low-temperature stress
GHK-Cu was not cytotoxic and did not induce any significant change in the expression levels of various skin irritation-related biomarkers
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