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Accumulating evidence has highlighted that APAP-induced oxidative stress and mitochondrial dysfunction are the fundamental factors in the pathogenesis of APAP-associated liver injury (ALI)
Several lines of evidence have suggested that impaired L-Glu homeostasis is linked to neuronal pathology and death in neurodegenerative diseases (NDDs), such as Alzheimers disease (AD) [10,11], Parkinsons disease (PD) [7,12,13], multiple sclerosis (MS) [14,15,16] amyotrophic lateral sclerosis (ALS) [17,18,19], and Huntingtons disease (HD) [20,21]
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