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This kinetic discrepancy has spurred investigations into rational combination strategies, particularly with BCL-2 inhibitors (e.g., venetoclax) or immune checkpoint modulators, to accelerate therapeutic efficacy and mitigate early treatment failure ( Likewise, the transcriptional rebound observed following BET inhibition in AML has been mechanistically linked to a compensatory feedback loop mediated by the histone acetyltransferase p300

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Mitochondria-targeted antioxidants for treatment of Parkinson's disease: preclinical and clinical outcomes

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