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Description
Disruption of the PDIA5-ATF6 axis restores sensitivity in imatinib-resistant leukemia cells, highlighting ATF6 as a therapeutically actionable target
The mechanism involves upregulation of tight junction proteins and mucosal blood flow via nitric oxide modulation (Seiwerth et al., 2018)
It is not considered likely to cause any undesirable effects during pregnancy
In addition, pathogen challenge elicited particularly dynamic responses of GSTF8: by 2 h after inoculation the corresponding transcript was already significantly up-regulated and the post-translational protein modifications detected were specific for incompatible interactions (Jones et al., 2004)
