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By further elucidating the pathogenic mechanism by which hepatic iron accumulation triggers ferroptosis through the c-Myc-Acsl4 regulatory axis, thereby accelerating the progression of metabolic-associated steatohepatitis (MASH), our research team identified a novel iron chelator, FOT1 (FerroTerminator1, formerly known as CN128), through drug screening, which can prevent and treat the onset and progression of MASH disease [117]

B.et al (2020)

Beyond vesicle content, the biogenesis and release of erythrocytes appears significantly influenced by oxidative stress, which is frequently reported in the peripheral blood of PD patients

Mechanisms, pathophysiology and currently proposed treatments of chronic obstructive pulmonary disease