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Collectively, these results suggest that the Ucp2 of lacrimal gland governs the SNS-controlled tear secretion in response to chronic dry eye stress
Xue et al., 2017) and attenuates high glucose-induced central nervous system injury in vitro through the activation of Nrf2 signaling, displaying protective effects against mitochondrial dysfunction-derived OS (Yang et al., 2017)
Structural homology modeling indicated a conserved structure among Egp38, EmMPK2, and H
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