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For instance, complement component 4A ( C4A ), located in the MHC locus, is highly associated with SCZ: specific structural variants and regulatory regions that increase the expression of C4A confer a greater risk for SCZ (Sekar et al., 2016)

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At the same time, LC is thought to down-regulate the expression of oxidative-stress-related genes such as Bax, Bcl-2, and caspase-3 [32]

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