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This distinctive type of cell death, reliant on iron-mediated phospholipid peroxidation (Wang et al., 2023), is intricately regulated by various cellular metabolic processes involving redox balance, iron homeostasis, mitochondrial function, as well as the metabolism of amino acids, lipids, and sugars, along with multiple associated signaling pathways relating to disease progression (Jiang X

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Structure-based insights into the catalytic power and conformational dexterity of peroxiredoxins