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Some papers described autophagy as a programmed cell death mechanism

It stimulates collagen, improves microcirculation, reduces inflammation, and supports hair follicle regeneration, making it one of the most advanced therapies in regenerative dermatology

GSH can be regenerated from GSSG by the enzyme glutathione reductase (GSR): NADPH reduces FAD present in GSR to produce a transient FADH-anion

These improvements were ascribed to SFs ability to attenuate LPS-induced pro-inflammatory cytokine production, restore synaptic protein expression, and normalize the BDNF-tropomyosin receptor kinase B (TrkB)-mechanistic target of rapamycin (mTOR) signaling in the hippocampus