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Collectively, these findings substantiate kaempferol's therapeutic promise as a safe, multifactorial candidate for both prevention and treatment of Alzheimer's disease by targeting oxidative stress, neuroinflammation, amyloid pathology, and neuronal dysfunction

This mechanistic link explains why oxidative stress can amplify SASP-mediated tissue damage, creating a self-reinforcing cycle of inflammation and cellular dysfunction.18 Senolytics reduce SASP not only by eliminating the senescent cells that produce high ROS but also by disrupting pro-survival signaling pathways that sustain SASP activity

To determine whether the directional segregation of the NZB mtDNAs in hematopoietic tissues was the result of immune surveillance, NZB-BALB heteroplasmic females were crossed onto nuclear backgrounds that lacked the Tap1, 2 m , or Rag1 genes