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It has been proposed that lipid accumulation may affect mitochondrial function, which in turn indirectly suppresses ACSL4 translocation, thereby exacerbating mitochondria-associated ferroptosis in the development of DbCM (Cai et al., 2024

Blood-brain barrier leakage is increased in parkinsons disease

Dehydroabietic acid improves nonalcoholic fatty liver disease through activating the Keap1/Nrf2-ARE signaling pathway to reduce ferroptosis

It has been argued that error signals conveyed by CFs suppress persistently inadequate PF-mediated input efficacies and the long-lasting modification between PF synapses on PC constitutes the substrate for motor learning in vestibulo-ocular reflex, eyeblink conditioning, and adaptive adjustments of hand movement [95, 96]