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The nuclear receptor signaling atlas: catalyzing understanding of thyroid hormone signaling and metabolic control

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Here we show that part of therapeutic effect of artificial tears supplemented with L-carnitine likely stems from its inhibition of TRPV1-induced keratocyte transdifferentiation into myofibroblasts

In fact, ALCAR is usually the preferred form in supplements for brain and nervous system support because it enters the brain quickly and is metabolized in mitochondria to L-carnitine, while its acetyl group is transferred to CoA to form acetyl-CoA