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This study was designed to systematically evaluate the therapeutic potential of GHK-Cu in a dextran sulfate sodium (DSS)-induced murine model of UC, with particular emphasis on elucidating its regulatory effects on the NAD-dependent deacetylase sirtuin-1 (SIRT1)/signal transducer and activator of transcription 3 (STAT3) signaling pathway
Nadph oxidase as a therapeutic target for oxalate induced injury in kidneys

By suppressing the secretion of pro-inflammatory cytokines such as NF-B, IL-8, and IL-6, NAC reduces the chemotactic migration of monocytes (Zhang et al., 2018)