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Brain 145, 17571762
Cmo se reconstituye y almacena el BPC-157
Using this mechanistic model of bile acids, we explain how abnormal bile acid physiology drives disease pathogenesis, emphasizing how ceramide synthesis may serve as a unifying pathogenic feature for cardiometabolic diseases
An increase in mitochondrial iron in murine models following ischemia/reperfusion injury, as well as in human hearts affected by ischemic heart disease, has been reported (Chang et al., 2016[74])
