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Here, we used a mouse model of chronic cutaneous leishmaniasis caused by the protozoan parasite Leishmania (L.) mexicana to investigate the function and transcriptomic signature of eosinophils in the skin

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These changes form the basis of the prooxidant state and lead to altered redox signaling related to glutathione deficiency and impaired expression and function of redox-sensitive transcriptional factors (Nrf2, NF- B, FoxO, etc.)

Sleep disruption in FM may impair NRF2-driven antioxidant responses, leading to increased oxidative stress and neuroinflammation, which further exacerbate pain and fatigue (30)