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Description
the load of free radicals
CSCs, with their self-renewal capacity and metabolic plasticity, drive tumor initiation, metastasis, and therapy resistance through intrinsic mechanisms and TME interactions, such as hypoxia and cytokine signaling [8, 9, 11, 13, 14, 18]
The benefit of HIPEC depends on the capacity of drug-exposed tumour cells to regenerate after drug washout
It should be noted, however, that modern mass spectrometry uses this overlap technique effectively
