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Due to hepatic metabolism of finasteride, precautions should be taken with patients with liver disease

KPV exerts its primary anti-inflammatory activity through direct intracellular penetration of immune cells and intestinal epithelial cells, where it suppresses NF-B transcription factor activity the master regulator of pro-inflammatory cytokine gene expression through a receptor-independent intracellular mechanism, resulting in downstream reduction of IL-6, IL-8, TNF-, and related inflammatory mediators

Patients should always consult with a qualified provider to understand which therapies are appropriate and legally available