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Microvasc Res 80: 394401, 2010
A deficit of VAChT, induced by SLC18A3 variants, are associated with the presynaptic congenital myasthenic syndrome, which is functionally characterized by electrodecrement on low-frequency repetitive stimulation and a prolonged period of postactivation exhaustion, as shown by electrophysiology studies conducted on congenital myasthenic syndrome affected patients [79]
Controlled study of the effects of dietary protein on blood pressure in normotensive humans
The modulation of this inflammatory microenvironment is a central focus in aging research