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To assess if cells with an undifferentiated or MES-like phenotype were affected, we performed immunostaining for the embryonic marker SOX9 (SRY-box transcription factor 9) and the marker of adrenergic (ADR) neuron differentiation TH (tyrosine hydrolase) 12

Although extrapolations from animal models to human MS patients must be made cautiously, these pre-clinical studies suggest that the KD may potentially reduce MS pathology and ameliorate associated symptoms

These changes can lower the threshold for pathogenic syn misfolding and aggregation, thereby exacerbating neurodegeneration [33]

[PubMed] [Google Scholar] 19.Ceaser EK, Moellering DR, Shiva S, et al