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Furthermore, mouse embryo fibroblast cells from GST null mice (deficient in both GSTP11 and P22) were less susceptible to TLK286 than their wild-type counterparts (Henderson et al ., 1998)

Additionally, the quantity of proinflammatory cytokines is directly correlated with the accumulation of A plaques in AD, partly due to reduced A clearance as well as increased astrocytic A production [147, 148]