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Aryl hydrocarbon receptor activation in chronic kidney disease: role of uremic toxins
AKT can directly phosphorylate Beclin1 at Ser234 and Ser295, promoting binding of Beclin1 to intermediate filaments and autophagy suppression (Mostowy, 2014
Immunostaining for AFP, ALBUMIN and A1AT proteins showed a significantly higher expression level in GF-HLCs in comparison to SM-HLCs (Figures 2H, I)
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