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Lead Researcher Spotlight Professor Ania M

Collectively, these studies demonstrate that aberrant LD accumulation in microglia represents a hallmark of dysfunctional, pro-inflammatory microglial states in the aging brain and is a consistent feature across neuroimmune-associated neurodegenerative pathologies

Figure 3 This action reduces the retention of Ly6G + neutrophils within the myocardium and downregulates the expression of pro-inflammatory cytokines like IL-6, TNF-, and IL-1, ultimately leading to improved cardiac functional parameters (148, 149)

Promyelocytic leukemia protein (PML) controls Listeria monocytogenes infection