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The cascade of events causing liver injury begins with the physiological dietary intake and intestinal absorption of copper, which cannot be adequately managed by patients with Wilson disease, ultimately resulting in severe copper dysregulation

however, as some of these transporters are cystineglutamate antiporters (Fig

Specifically, the T alleles of rs8078340 and rs2779249, along with G alleles of rs2779248, lead to higher NO production

Various studies have validated the anti-inflammatory and anti-fibrotic potential of alkaloids (Liu et al., 2023), polysaccharides, flavonoids, peptides, terpenoids, and polyphenols (Midgley et al., 2020), influencing the synthesis and secretion of pro-fibrotic factors (Li et al., 2017a), and regulating ECM synthesis and degradation, thereby offering therapeutic benefits for an anti-fibrotic treatment